London's clean air zone rapidly restores children's stunted lung growth

Children in polluted areas experienced stunted lung development and increased disease risk; one child, Ella Adoo-Kissi-Debrah, died from asthma with air pollution listed as a cause.
The speed of catch-up was surprising and impressive.
A researcher's reaction to discovering London children's stunted lungs had accelerated their growth to match peers in cleaner air.
Mark

What made the researchers so surprised by these results?

Mimi

They expected to see improvement, sure, but the speed was the shock. Children's lungs normally grow year by year at a predictable rate. What happened in London was acceleration—the stunted lungs didn't just start growing normally again, they grew faster to catch up. That's not how biology usually works.

Mark

So the pollution had actually slowed their growth, not just made them smaller?

Mimi

Exactly. The pollution was actively suppressing how fast their lungs developed. Once the air got cleaner, that suppression lifted and growth rebounded. It's like removing a weight that was holding them back.

Mark

Why are children so much more vulnerable to this than adults?

Mimi

Their lungs are still building themselves. An adult's lungs are finished growing, so pollution damages them but doesn't stunt development the way it does in kids. Plus children breathe faster and play closer to the ground where exhaust fumes sit thickest. They're getting a higher dose at a critical moment.

Mark

The study compared London to Luton. Why not just look at London before and after?

Mimi

Because you need a control group. Air pollution was improving everywhere in Britain during those five years, not just in London. By comparing London to Luton, they could see how much of the improvement was specifically from Ulez and how much was just the general trend. That's what made the comparison so valuable.

Mark

Does this mean Ulez solved the problem?

Mimi

No. The researchers were clear about that. Even after the improvements, pollution in both cities still exceeds what the World Health Organization says is safe. Ulez helped, significantly, but there's still a long way to go.

Mark

What happens to these children now? Does the lung improvement stick?

Mimi

That's the hope, and the evidence suggests it should. The researchers say the improvements are likely to have lifelong health benefits. But that's a question that will take decades to fully answer.

  • More than 3,400 London and Luton primary school children tracked for five years starting in 2019
  • Clinically impaired lung capacity in London fell from 14% to 9% over the study period
  • Children in London's ULEZ showed accelerated lung growth to match less-polluted areas within four years
  • Ella Adoo-Kissi-Debrah, age 9, became the first UK person with air pollution listed as cause of death in 2020

Children in London's ULEZ showed accelerated lung growth to match less-polluted areas within four years, with clinically impaired lung capacity dropping from 14% to 9%. Air pollution stunts lung development in children, increasing risks of asthma, heart disease, and premature death; children are especially vulnerable due to developing immune systems.

A five-year study of London schoolchildren shows lung capacity recovered significantly after the Ultra Low Emission Zone reduced air pollution, with researchers describing the rapid improvement as 'stunning' and suggesting lifelong health benefits.

In 2019, London introduced a policy that would become a natural experiment in children's health. The Ultra Low Emission Zone, or Ulez, required older, more polluting vehicles to pay a daily charge to drive in central London—a measure that would later expand across the entire city. What researchers found in the years that followed surprised even the scientists conducting the work: children whose lungs had been stunted by years of breathing London's dirty air began to recover with remarkable speed.

A team led by Queen Mary University of London tracked more than 3,400 primary school children over five years, starting in the year before Ulez took effect. They measured the same children annually, testing how much air they could forcibly exhale in a single second after taking a deep breath. They also compared London children to a control group of similar age and background in Luton, a less polluted city with a comparable mix of pollutants. The initial results were stark: London children's lungs were measurably smaller in capacity than their Luton peers.

But something unexpected happened. By the end of the study period, the gap had closed. The London children's lungs had grown faster than normal—accelerating to catch up with the Luton group until their lung capacity reached nearly identical levels. "I was absolutely stunned when I first saw the results," said Prof Chris Griffiths, a senior author on the study. "The speed of catch up was surprising and impressive." In practical terms, the improvements meant London children could now run without getting winded as easily, or blow out the same number of candles as their peers in cleaner air.

The scale of the shift was significant. The proportion of London children with clinically impaired lung capacity—a measure suggesting lung damage that causes coughs or breathlessness—fell from 14 percent to 9 percent over the study period. In Luton, where smaller-scale pollution measures had been implemented, the figure dropped from 9 percent to 7 percent. The researchers also measured nitrogen dioxide exposure, the harmful gas that comes primarily from motor vehicles. Children in London experienced faster reductions in their exposure to this pollutant than children in Luton, suggesting the lung improvements were directly tied to the air quality gains from Ulez.

The findings matter because air pollution damages developing lungs in ways that can persist for life. Children are especially vulnerable—their lungs and immune systems are still forming, and when they play outside, they breathe closer to the ground where exhaust fumes concentrate. Stunted lung growth increases the risk of asthma, heart disease, diabetes, and premature death. The case of Ella Adoo-Kissi-Debrah, a nine-year-old Londoner who died from an asthma attack in 2013, underscored the stakes. In 2020, she became the first person in the UK to have air pollution officially listed as a cause of death.

Independent researchers offered measured praise for the work while noting important caveats. Prof Anna Hansell at the University of Leicester, who was not involved in the study, called the findings "carefully conducted" and noted that the comparison between London and Luton was particularly valuable because it controlled for general air pollution improvements happening across the country—and crucially, it showed the findings were not simply an artifact of the Covid-19 pandemic, which occurred during the study period. Other experts suggested that increased walking and cycling to school in London after Ulez came in might have contributed to some of the health gains.

Yet the researchers themselves cautioned against complacency. Dr Helen Wood, the study's lead author, noted that air pollution in both London and Luton remains above World Health Organization guideline levels. "There is still work to be done," she said. Griffiths added that the findings carry global weight: more than a billion children live in cities worldwide, most in polluted environments. "That's why these are important findings with global relevance," he said. The study, published in the Lancet Public Health, involved researchers from universities across the UK and the United States, and it offers what scientists believe is the strongest evidence yet that ambitious clean air zones can reverse some of the damage pollution inflicts on developing bodies.

The speed of catch up in lung capacity in the London group was surprising and impressive. This shows an ambitious clean air zone can drive pollution levels down, rapidly restoring children's stunted lung growth.
— Prof Chris Griffiths, Queen Mary University of London
Air pollution in both London and Luton remains above WHO guideline levels, so there is still work to be done.
— Dr Helen Wood, lead author, Queen Mary University of London
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